Development of Rif-resistance in wild type Mtb H37Rv but not in Δ-tgs1 mutant upon application of multiple-stress; complementation restores Rif-resistance.
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https://figshare.com/articles/dataset/_Development_of_Rif_resistance_in_wild_type_Mtb_H37Rv_but_not_in_tgs1_mutant_upon_application_of_multiple_stress_complementation_restores_Rif_resistance_/564321
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Mtb cultures at 0, 9 or 18 days of multiple-stress were treated with Rif (5 µg/ml) or INH (0.8 µg/ml) for 5 days. Viable bacilli were enumerated by the cfu count method and compared with controls not subjected to antibiotic treatment. WT-H37RV, Mtb H37RV, Δ-tgs1, tgs1 deletion mutant of WT-H37Rv, C-Δ-tgs1, complemented tgs1 mutant. Rif, Rifampicin; INH, Isoniazid.
创建时间：
2015-12-02
