Identification and targeting of the developmental blockade in extranodal natural killer/T cell lymphoma

NIAID Data Ecosystem2026-05-01 收录

皮肤T细胞淋巴瘤分子病理机制

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE169646 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

创建时间：

2023-07-13

RNA sequencing of whole spleens infiltrated with murine peripheral T-cell lymphoma - time course. RNA sequencing of whole spleens from control mice and mice with peripheral T-cell lymphoma days 7, 14 and 21 after implant

皮肤T细胞淋巴瘤分子病理机制

DNA损伤反应抑制治疗

Peripheral T-cell lymphoma (PTCL) represents a rare group of heterogenous diseases in urgent need of effective treatments. A scarcity of disease-relevant preclinical models has considerably hindered r

NIAID Data Ecosystem90

Gene expression profiling of CTCL cell line Hut78 via IKZF2 knockdown by RNA-seq

皮肤T细胞淋巴瘤分子病理机制

基因功能分析

IKZF2 is an important nuclear matrix protein and plays a pivotal role in T cell development and differentiation, while its expression and function in Cutaneous T cell lymphoma (CTCL) remain ambiguous.

NIAID Data Ecosystem80

Table_1_MicroRNA and circRNA Expression Analysis in a Zbtb1 Gene Knockout Monoclonal EL4 Cell Line.xls

皮肤T细胞淋巴瘤分子病理机制

植物非编码RNA表达调控

Zinc finger and BTB domain containing 1(Zbtb1) is a transcriptional suppressor protein, and a member of the mammalian Zbtb gene family. Previous studies have shown that Zbtb1 is essential for T-cell d

NIAID Data Ecosystem50

Expression data from Dnmt3a-deficient and control mouse MYC induced T-cell lymphomas. Mus musculus

皮肤T细胞淋巴瘤分子病理机制

甲基化与基因表达关联

Dnmt3a catalyzes DNA methylation of gDNA, which contributes to the transriptional regulations of genes and genomic stability. Overall design: Dnmt3a was deleted in the hematopoietic compartment in the

NIAID Data Ecosystem80

Expression data from Dnmt3a-deficient and control mouse MYC induced T-cell lymphomas

皮肤T细胞淋巴瘤分子病理机制

表观遗传与肿瘤

Dnmt3a catalyzes DNA methylation of gDNA, which contributes to the transriptional regulations of genes and genomic stability. Dnmt3a was deleted in the hematopoietic compartment in the presence of MYC

NIAID Data Ecosystem70

Identification and targeting of the developmental blockade in extranodal natural killer/T cell lymphoma

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