Methylome Analysis of FF HPV+ and HPV- HNSCC samples

NIAID Data Ecosystem2026-03-11 收录

头颈癌甲基化组分析

HPV感染表观遗传学

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE38263 数据链接链接失效反馈

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资源简介：

The analysis of the 6 methylomes by MeDIP-Seq comprise 3 HPV+ HNSCC samples and 3 HPV- HNSCC samples Analysis of the 6 methylomes, comprising 3 HPV+ HNSCC samples and 3 HPV- HNSCC samples

创建时间：

2019-05-15

Methylome Analysis of FF HPV+ and HPV- HNSCC samples. Homo sapiens

头颈部鳞状细胞癌甲基化

HPV感染表观遗传学

The analysis of the 6 methylomes by MeDIP-Seq comprise 3 HPV+ HNSCC samples and 3 HPV- HNSCC samples Overall design: Analysis of the 6 methylomes, comprising 3 HPV+ HNSCC samples and 3 HPV- HNSCC samp

NIAID Data Ecosystem40

Correlation between High and Low Risk HPV status and methylation fraction means at differentially methylated region (DMRs) of PEG3.

HPV感染表观遗传学

甲基化分析

High Risk HPV - 16,18, 31, 33, 35, 39, 45, 51, 52, 56, 58, 59, 66, 68.Low/Other Risk HPV – 6, 11, 26,40, 42, 55, 61, 62, 69, 70, 72, 73, 81, 82, 83, 84.

Figshare2015-12-02 更新30

Aberrant methylation-mediated silencing of microRNAs contributes to HPV-induced anchorage independence [methylation-Illumina]

HPV感染表观遗传学

肿瘤抑制miRNA甲基化

Cervical cancer and a subset of anogenital and head-and-neck carcinomas are caused by persistent infection with high-risk types of the human papillomavirus (hrHPV). Early stages of hrHPV-induced carci

NIAID Data Ecosystem20

Supplementary Material for: Analysis of p16 CDKN2A Methylation and HPV-16 Infection in Oral Mucosal Dysplasia

口腔癌机制

HPV感染表观遗传学

Objective: The purpose of this study was to investigate the relationship between p16CDKN2A methylation and epithelial dysplasia (ED). We also evaluated the expressions of proteins related to methylati

NIAID Data Ecosystem30

Table_2_The antigen processing-associated transporter gene polymorphism: Role on gene and protein expression in HPV-infected pre-cancerous cervical lesion.xlsx

HPV感染表观遗传学

抗原处理转运蛋白多态性

Human papillomavirus (HPV) is the major pathogen for cervical lesions. The evasion mechanism of the immune response and persistence of HPV infection can be influenced by polymorphisms (SNPs) in genes

NIAID Data Ecosystem20

Methylome Analysis of FF HPV+ and HPV- HNSCC samples

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