ATR inhibition enhances 5-fluorouracil sensitivity independent of non-homologous end-joining and homologous recombination repair pathway
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https://www.ncbi.nlm.nih.gov/sra/DRP006043
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资源简介：
To investigate the nature of the cell cycle arrest involved in the response to 5-FU treatment, we analyzed genome-wide mRNA by bulk RNA sequencing. The present study used SAS and HSC3 human oral squamous cell carcinoma cell lines.
创建时间：
2020-05-21
