NFIB dependent and independent mechanisms of metastasis in small cell lung cancer
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2024-02-09
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NFIB dependent and independent mechanisms of metastasis in small cell lung cancer [ATAC-seq]. NFIB dependent and independent mechanisms of metastasis in small cell lung cancer [ATAC-seq]
Molecular characterization of lung tumors and liver metastases from SCLC mouse models with and without Nfib knockout. We performed gene expression profiling analysis using data obtained from ATAC-seq
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KMT2C Loss Promotes Small Cell Lung Cancer Metastasis through DNMT3A-mediated Epigenetic Reprogramming. KMT2C Loss Promotes Small Cell Lung Cancer Metastasis through DNMT3A-mediated Epigenetic Reprogramming
Small cell lung cancer (SCLC) is notorious for its early and frequent metastases, which contribute to it as a recalcitrant malignancy. To understand the molecular mechanisms underlying SCLC metastasis
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Nfib promotes Metastasis through a Widespread Increase in Chromatin Accessibility [ATAC-seq]
We describe Nfib as an important regulator of chromatin accessibility in Small cell lung cancer (SCLC). Overall design: ATAC seq was performed on [1] ex vivo samples from primary tumors and metastases
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KMT2C Loss Promotes Small Cell Lung Cancer Metastasis through DNMT3A-mediated Epigenetic Reprogramming
Small cell lung cancer (SCLC) is notorious for its early and frequent metastases, which contribute to it as a recalcitrant malignancy. To understand the molecular mechanisms underlying SCLC metastasis
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NFIB dependent and independent mechanisms of metastasis in small cell lung cancer [RNA-seq]. NFIB dependent and independent mechanisms of metastasis in small cell lung cancer [RNA-seq]
Molecular characterization of lung tumors and liver metastases from SCLC mouse models with and without Nfib knockout. We performed gene expression profiling analysis using data obtained from RNA-seq o
NIAID Data Ecosystem20
