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File S1 - CNF1 Increases Brain Energy Level, Counteracts Neuroinflammatory Markers and Rescues Cognitive Deficits in a Murine Model of Alzheimer's Disease

NIAID Data Ecosystem2026-03-07 收录

阿尔茨海默病神经炎症

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数据链接：

https://figshare.com/articles/dataset/_CNF1_Increases_Brain_Energy_Level_Counteracts_Neuroinflammatory_Markers_and_Rescues_Cognitive_Deficits_in_a_Murine_Model_of_Alzheimer_s_Disease_/709416 数据链接链接失效反馈

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资源简介：

Contains Tables S1-S20. Summary of the statistical analysis. (DOC)

应用场景：

创建时间：

2013-05-30

阿尔茨海默病神经炎症

干细胞可塑性

The immune response is an important determinant of the plasticity and neurogenic capacity of neural stem cells (NSCs) upon amyloid-beta42 (Aβ42) toxicity in Alzheimer’s disease (AD). However, the dire

NIAID Data Ecosystem70

Bradykinin promotes immune responses in differentiated embryonic neurospheres carrying APP swe and PS1 dE9 mutations. Bradykinin promotes immune responses in differentiated embryonic neurospheres carrying APP swe and PS1 dE9 mutations

阿尔茨海默病神经炎症

基因表达调控

Neural stem cells (NSCs) can be cultivated from developing brains, reproducing many of the processes that occur during neural development. They can be isolated from a variety of animal models, such as

NIAID Data Ecosystem40

β-Amyloid precursor protein transgenic mice that harbor diffuse Aβ deposits but do not form plaques show increased ischemic vulnerability: Role of inflammation

阿尔茨海默病神经炎症

脑缺血损伤易感性

β-amyloid (Aβ), derived form the β-amyloid precursor protein (APP), is important for the pathogenesis of Alzheimer's disease (AD), which is characterized by progressive decline of cognitive functions,

PubMed Central2002-01-29 更新30

Table1.xlsx

阿尔茨海默病神经炎症

信号通路

Alzheimer's disease (AD) is a major neurodegenerative disease and is one of the most common cause of dementia in older adults. Among several factors, neuroinflammation is known to play a critical role

NIAID Data Ecosystem60

Innate immune signaling by ZBP1-RIPK1 axis drives neuroinflammation in Alzheimer's disease

阿尔茨海默病神经炎症

先天免疫信号通路

Z-DNA is a non-canonical structure capable of activating innate immune signaling through Z-DNA-binding protein 1 (ZBP1). However, the functional significance of ZBP1-mediated detection of Z-DNA in Alz

NIAID Data Ecosystem30

File S1 - CNF1 Increases Brain Energy Level, Counteracts Neuroinflammatory Markers and Rescues Cognitive Deficits in a Murine Model of Alzheimer's Disease

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