Additional file 9 of Smarcad1 mediates microbiota-induced inflammation in mouse and coordinates gene expression in the intestinal epithelium

Additional file 9: Table S8. Differential chromatin accessibility MACS-peaks (300 bp fragment size) identified with the EdgeR test (cut-off FDR < 0.00001) on comparison of ATAC-seq datasets from WT and Smarcad1-KO (Villin-cre mediated) small intestinal whole crypt isolates. Table contents: MACS-peak chromosomal position, closest gene (5 kbp cut-off) with distance and annotation, EdgeR FDR (Benjamini-Hochberg corrected) and quantitation (n = 3, log2, read counts normalized to largest data store) in WT/KO datasets by sample.补充文件9：表S8。本表格收录了通过EdgeR检验（错误发现率截断阈值FDR < 0.00001），对比野生型（Wild Type, WT）与Smarcad1基因敲除型（Smarcad1-KO，经Villin-cre重组酶介导构建）的小肠全隐窝分离物的转座酶可及性测序（Assay for Transposase-Accessible Chromatin using sequencing, ATAC-seq）数据集后，以300 bp片段大小为参数鉴定得到的差异染色质可及性MACS峰（MACS-peaks）。表格包含以下内容：MACS峰的染色体位置、上下游5 kbp截断阈值范围内的最近基因及其对应距离与功能注释、经Benjamini-Hochberg校正后的EdgeR检验错误发现率（FDR），以及各样本野生型/敲除型数据集的定量数据（n=3，采用log2转换，读计数已归一化至最大数据集）。