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Amyotrophic lateral sclerosis. Homo sapiens

NIAID Data Ecosystem2026-03-06 收录

ALS相关基因鉴定

神经退行性分子机制

数据链接：

https://www.ncbi.nlm.nih.gov/bioproject/PRJNA87835 数据链接链接失效反馈

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资源简介：

Identification of amyotrophic lateral sclerosis (ALS) associated genes. Post mortem spinal cord grey matter from sporadic and familial ALS patients compared with controls. Keywords: other

应用场景：

创建时间：

2003-11-14

衰老相关蛋白

神经退行性分子机制

The attachment of Post-Translational Modifications (PTMs) to proteins plays key roles in the regulation of the activity and stability of various proteins. Here we utilized the nematode Caenorhabditis

NIAID Data Ecosystem40

Additional file 3 of Cytokine enrichment in deep cerebellar nuclei is contributed by multiple glial populations and linked to reduced amyloid plaque pathology

神经退行性分子机制

单细胞RNA测序

Additional file 3: Supplementary Data for snRNAseq and RNAseq experiments.

Figshare2024-09-11 更新10

TDP-43表观遗传学

神经退行性分子机制

This is the summarized excel file containing the original information for all experiment-based figures and supplementary figures in our manuscript "Age-related demethylation of the TDP-43 autoregulato

Figshare2021-01-27 更新10

Global gene profiling of (-)-Epigallocatechin-3-gallate (EGCG)-induced neuronal death. Mus musculus

神经退行性分子机制

药物神经保护

Alzheimer’s disease (AD) is the most common form of adult-onset dementia with severe intellectual deterioration and is characterised by the accumulation of the amyloid-β (Aβ) peptides and the presence

NIAID Data Ecosystem30

Brain Extracellular Matrix implications in multiple neurological disorders revealed through a meta-analysis of transcriptional changes

神经退行性分子机制

元转录组分析

Authors: Hagit Sadis1*, Wote Amelo Rike1*, David Peles1, Yara Hussein1, Tchelet Stern1, Irit Sagi2, Shani Stern1* Affiliations: 1Sagol Department of Neurobiology, Faculty of Natural Sciences, Univer

Zenodo2025-11-11 更新10

Amyotrophic lateral sclerosis. Homo sapiens

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