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single nucleus RNA-seq of early development in P. miniata and S. purpuratus. single nucleus RNA-seq of early development in P. miniata and S. purpuratus

NIAID Data Ecosystem2026-05-01 收录

单细胞转录组学

早期发育比较

数据链接：

https://www.ncbi.nlm.nih.gov/bioproject/PRJNA1002357 数据链接链接失效反馈

官方服务：

资源简介：

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

应用场景：

创建时间：

2023-08-04

单细胞转录组学

马哮喘免疫病理学

The transcriptomic profile of a cell population can now be studied at the cellular level using single-cell mRNA sequencing (scRNA-seq). This novel technique provides the unprecedented opportunity to e

NIAID Data Ecosystem20

ScRNA-Seq of Tongue Tissues in Chronic Hyperplastic Candidiasis

单细胞转录组学

口腔免疫病理

The first single-cell RNA sequencing (scRNA-seq) analysis of CHC-affected tongue tissues were performed to reveal its microenvironmental changes and immunological etiology.

NIAID Data Ecosystem00

Additional Data from: Monocyte-derived transcriptome signature indicates antibody-dependent cellular phagocytosis as a potential mechanism of vaccine-induced protection against HIV-1

HIV-1疫苗免疫机制

单细胞转录组学

Raw (non-normalized) gene expression and antibody feature counts for RV306 single cell object in "Monocyte-derived transcriptome signature indicates antibody-dependent cellular phagocytosis as the pri

NIAID Data Ecosystem00

Data_Sheet_2_Infection of Monocytes From Tuberculosis Patients With Two Virulent Clinical Isolates of Mycobacterium tuberculosis Induces Alterations in Myeloid Effector Functions.zip

牛结核病与副结核病免疫机制

单细胞转录组学

Monocytes play a critical role during infection with Mycobacterium tuberculosis (Mtb). They are recruited to the lung, where they participate in the control of infection during active tuberculosis (TB

NIAID Data Ecosystem20

Table_2_Single cell RNA sequencing analysis of mouse cochlear supporting cell transcriptomes with activated ERBB2 receptor indicates a cell-specific response that promotes CD44 activation.DOCX

耳蜗细胞再生

单细胞转录组学

Hearing loss caused by the death of cochlear hair cells (HCs) might be restored through regeneration from supporting cells (SCs) via dedifferentiation and proliferation, as observed in birds. In a pre

NIAID Data Ecosystem00