Frequency (that is, proportion of incidences) of each of the six types of cell shape division outcome over the entire cell lineages analysed for the sixteen curved mutants and the wild-type.

Measurements are based on the phenotype of progenitors and progeny before cell septation. 1–6 are represented in Fig. 1D. The columns on the right indicate the measured penetrance at septation (left) and predicted penetrance at septation (by the Markovian model) based on the six frequencies (right), for each mutant. Importantly, the measured penetrances and the frequencies come from independent biological experiments. For all strains other than alp14Δ and pom1Δ both penetrances were within ±5% difference. This likely due to the fact that alp14Δ cells often do not undergo division (not shown) and that pom1Δ cells divide highly asymmetrically [18], both features which are not accounted for by our model.Frequency (that is, proportion of incidences) of each of the six types of cell shape division outcome over the entire cell lineages analysed for the sixteen curved mutants and the wild-type.