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Neuroblastoma cell lines treated with DAC (2'-deoxy-5-azacytidine), a DNA-methylation inhibitor

ChEBI2012-09-13 更新2026-07-21 收录

母细胞瘤表观遗传学

DNA甲基化抑制剂作用

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-31229 数据链接链接失效反馈

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Neuroblastoma cell lines treated with DAC (2'-deoxy-5-azacytidine), a DNA-methylation inhibitor

应用场景：

创建时间：

2012-09-13

母细胞瘤表观遗传学

5hmC修饰分析

Additional file 8: Table S7. Summary of 5hmC gain and loss regions in SmoA1-MBs.

Figshare2024-02-20 更新70

DNA methylome evolution and reprogramming in recurrent glioblastoma

母细胞瘤表观遗传学

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Recurrent glioblastoma (GBM) has a grim prognosis, though MGMT promoter methylation and IDH mutation provide a significant survival advantage. The product of IDH mutation, 2-hydroxyglutarate, increas

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Differential gene expression of neuroblastoma cells stably expressing doxycycline-inducible DOT1L small hairpin RNA. Homo sapiens

母细胞瘤表观遗传学

DOT1L敲低效应

We were interested in studying the role of DOT1L-mediated H3K79 methylation on N-Myc target gene transcription in MYCN-induced neuroblastoma. We aimed to determine whether knocking down DOT1L in MYCN-

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MLL5 Orchestrates a Cancer Self-Renewal State by Repressing the Histone Variant H3.3 and Globally Reorganizing Chromatin [expression]

母细胞瘤表观遗传学

干细胞自我更新

Mutations in the histone 3 variant H3.3 have been identified in one-third of pediatric glioblastomas (GBMs), but not in adult tumors. Here we show that H3.3 is a dynamic determinant of functional prop

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EGFR Mutation Promotes Glioblastoma Through Epigenome and Transcription Factor Network Remodeling

母细胞瘤表观遗传学

EGFR突变致癌机制

Epidermal Growth Factor Receptor (EGFR) gene amplification and mutations are the most common oncogenic events in Glioblastoma (GBM), but the mechanisms by which they promote aggressive tumor growth ar

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Neuroblastoma cell lines treated with DAC (2'-deoxy-5-azacytidine), a DNA-methylation inhibitor

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