Doctoral Thesis Presentation.pdf

Rationale for the interaction of protein kinase complexes AMPK, LKB1, and PGC-1a by acute hypoxia induced by blood flow restriction training in individuals with fibromyalgia.

纤维肌痛（fibromyalgia）患者在血流限制训练（blood flow restriction training）诱导的急性缺氧（acute hypoxia）状态下，蛋白激酶复合物（protein kinase complexes）AMP活化蛋白激酶（AMPK）、肝激酶B1（LKB1）与过氧化物酶体增殖物激活受体γ辅激活因子1α（PGC-1α）之间相互作用的理论依据。