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Data for: Cryptotanshinone ameliorates the pathogenesis of systemic lupus erythematosus by blocking T cell proliferation

NIAID Data Ecosystem2026-03-11 收录

系统性红斑狼疮药物机制

T细胞免疫抑制

数据链接：

https://data.mendeley.com/datasets/3xnbnmzw8j 数据链接链接失效反馈

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资源简介：

These contained all data appearing in this paper

本数据集包含本文中出现的全部数据

应用场景：

创建时间：

2019-06-07

弓形虫毒力因子ROP16

T细胞免疫抑制

The ability of Toxoplasma gondii to inject the rhoptry kinase ROP16 into host cells results in the activation of the transcription factors STAT3 and STAT6, but it is unclear how these events impact in

NIAID Data Ecosystem60

Structure and Function of a Fungal Adhesin that Binds Heparin and Mimics Thrombospondin-1 by Blocking T Cell Activation and Effector Function

真菌致病性

T细胞免疫抑制

Blastomyces adhesin-1 (BAD-1) is a 120-kD surface protein on B. dermatitidis yeast. We show here that BAD-1 contains 41 tandem repeats and that deleting even half of them impairs fungal pathogenicity.

NIAID Data Ecosystem40

Data_Sheet_1_iCa2+ Flux, ROS and IL-10 Determines Cytotoxic, and Suppressor T Cell Functions in Chronic Human Viral Infections.pdf

慢性感染机制

T细胞免疫抑制

Exhaustion of CD8+ T cells and increased IL-10 production is well-known in chronic viral infections but mechanisms leading to loss of their cytotoxic capabilities and consequent exhaustion remain uncl

NIAID Data Ecosystem30

IL-12-induced immune suppressive deficit during CD8+ T-cell differentiation

T细胞免疫抑制

自身免疫疾病治疗

Autoimmune diseases are characterized by regulatory deficit in both the CD4+ and CD8+ T-cell compartments. We have shown that CD8+ T-cells associated with acute relapse of multiple sclerosis are signi

NIAID Data Ecosystem40

Suppression of antitumor immunity by the oncometabolite 2-hydroxyglutarate. Homo sapiens

肿瘤代谢免疫

T细胞免疫抑制

Isocitrate dehydrogenase (IDH) mutations, a hallmark of gliomagenesis, result in the production of the oncometabolite R-2-hydroxyglutarate (R-2-HG) which is thought to promote tumorigenesis via DNA me

NIAID Data Ecosystem40

Data for: Cryptotanshinone ameliorates the pathogenesis of systemic lupus erythematosus by blocking T cell proliferation

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