Defective removal of ribonucleotides from DNA promotes systemic autoimmunity

ChEBI2014-12-15 更新2026-07-21 收录

DNA修复缺陷

自身免疫病机制

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-63755 数据链接链接失效反馈

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Defective removal of ribonucleotides from DNA promotes systemic autoimmunity

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2014-12-15

Gut dysbiosis contributes to the imbalance of Treg and Th17 cells in Graves’ disease patients by propionic acid

自身免疫病机制

肠道菌群免疫调节

Supplementary material for manuscript "Gut dysbiosis contributes to the imbalance of Treg and Th17 cells in Graves’ disease patients by propionic acid".

Figshare2020-07-17 更新70

Prmt5 deficiency inhibits CD4+T cells migration through Klf2-S1pr1 and ameliorates EAE disease (scATAC-seq)

自身免疫病机制

细胞迁移

Protein arginine methyltransferase 5 (Prmt5) is the major type II methyltransferase catalyzing symmetric dimethylation, regulates cell development, homeostatic and disease processes. Prmt5 inhibition

NIAID Data Ecosystem40

Mutational_Signatures_in_the_YKO_collection. Mutational_Signatures_in_the_YKO_collection

基因组突变特征

DNA修复缺陷

This project is being carried out through a Strategic Award from the Wellcome Trust to Steve Jackson, Mike Stratton, Peter Campbell, Serena Nik-Zainal and others. This grant is to fund work aimed at i

NIAID Data Ecosystem30

Diabetes-associated breast cancer has a distinct transcriptome and metabolome and shows DNA repair deficiency

糖尿病相关乳腺癌组学

DNA修复缺陷

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

NIAID Data Ecosystem50

Ability of V. cholerae mutants defective in DNA repair pathways to colonize the infant mouse intestine in competition with the parental strain (WT).

肠道菌群定植

DNA修复缺陷

aThe competitive index is the ratio of mutant to parental (WT) cfu in the small intestine post infection divided by the input ratio of mutant to parental (WT) cfu. The average and standard error of 5–

NIAID Data Ecosystem30

Defective removal of ribonucleotides from DNA promotes systemic autoimmunity

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