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Pancreatic Beta Cell Enhancers Regulate Rhythmic Transcription of Exocyst Triggering and Diabetes

ChEBI2015-11-10 更新2026-07-12 收录

糖尿病分子机制

细胞转录调控

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-70961 数据链接链接失效反馈

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Pancreatic Beta Cell Enhancers Regulate Rhythmic Transcription of Exocyst Triggering and Diabetes

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2015-11-10

细胞转录调控

免疫基因组学

In an effort to study gene expression modulated by RORɣt and RORα in human Th17 cells, global gene expression of human CD4 T cells activated under Th17 skewing conditions was profiled by RNA sequencin

NIAID Data Ecosystem70

RNA-seq of isolated pancreatic islets from beta-cell-specific Jazf1 knock-out mice

糖尿病分子机制

胰岛β细胞功能基因组学

Genome-wide association studies (GWAS) have identified several susceptibility loci for T2D. Among these loci, juxtaposed with another zinc finger protein 1 gene (JAZF1) has been shown as one of the to

NIAID Data Ecosystem30

Disturbance of lipid and glucose metabolism induce beta cell dedifferentiation in Ins-1 cell line

胰岛β细胞

糖尿病分子机制

In this study, we tried to clarify whether abnormal glucose and lipid metabolism can lead to beta cell dedifferentiation in vitro , and whether there is the role of transcription factor Foxo1 in this

NIAID Data Ecosystem30

The Hippo Pathway Prevents YAP/TAZ–Driven Hypertranscription and Controls Neural Progenitor Number [ChIP-seq]. The Hippo Pathway Prevents YAP/TAZ–Driven Hypertranscription and Controls Neural Progenitor Number [ChIP-seq]

细胞转录调控

Hippo信号通路

The Hippo pathway controls the activity of YAP/TAZ transcriptional coactivators through a kinase cascade. Despite the critical role of this pathway in tissue growth and tumorigenesis, it is not fully

NIAID Data Ecosystem60

Quantitative proteomic analysis of adipose tissue in obese or obese T2D patients(TMT method)

脂肪组织蛋白质组学

糖尿病分子机制

To find out the regulating pathways of AIP1 in Type 2 Diabetes, we performed a proteomic array of omental adipose tissue between obese T2D and obese patients.

NIAID Data Ecosystem30

Pancreatic Beta Cell Enhancers Regulate Rhythmic Transcription of Exocyst Triggering and Diabetes

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