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Suppl. Table 1 from Upregulation of AKT3 Confers Resistance to the AKT Inhibitor MK2206 in Breast Cancer

Figshare2016-08-01 更新2026-04-28 收录

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https://figshare.com/articles/dataset/Suppl_Table_1_from_Upregulation_of_AKT3_Confers_Resistance_to_the_AKT_Inhibitor_MK2206_in_Breast_Cancer/22507506 数据链接链接失效反馈

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Table showing differentially expressed genes in T47D MK2206-resistant cells.

展示T47D MK2206耐药细胞中差异表达基因的表格

应用场景：

创建时间：

2016-08-01

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癌症表观遗传学

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Aberrant DNA-methylation at CpG dinucleotides is a hallmark of cancer and is associated with the emergence of resistance to anti-cancer treatment, though molecular mechanisms and biological signifi-

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DataSheet2_Exogenous proline enhances susceptibility of NSCLC to cisplatin via metabolic reprogramming and PLK1-mediated cell cycle arrest.xlsx

化疗耐药机制

代谢重编程

The occurrence of cisplatin resistance is still the main factor limiting the therapeutic effect of non-small cell lung cancer (NSCLC). It is urgent to elucidate the resistance mechanism and develop no

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Unveiling alterations of epigenetic modifications and chromatin architecture leading to lipid metabolic reprogramming during the evolutionary trastuzumab adaptation of HER2-positive breast cancer [Cut & Tag]. Unveiling alterations of epigenetic modifications and chromatin architecture leading to lipid metabolic reprogramming during the evolutionary trastuzumab adaptation of HER2-positive breast cancer [Cut & Tag]

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表观遗传调控

Secondary trastuzumab resistance represents an evolutionary adaptation of HER2-positive breast cancer during anti-HER2 treatment. Most current studies have uncovered the alternations of HER2 and assoc

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Suppl. Table 1 from Upregulation of AKT3 Confers Resistance to the AKT Inhibitor MK2206 in Breast Cancer

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