All data

IV relationships from NMDA receptor and AMPA/kainate currents from hippocampal Schaffer-CA1 synapses from sham and sound stimulated animals.-Data from AMPA/kainate EPSCs recorded at -80 mV from Schaffer-CA1 synapses in hippocampal pyramidal neurons from rats sham and sound stimulated animals.-Data from NMDA EPSCs recorded at 70 mV from Schaffer-CA1 synapses in hippocampal pyramidal neurons from rats sham and sound stimulated animals. All glutamatergic currents were recorded in the presence of picrotixi.-Data from GABAergic spontaneous IPSCs (in the presence of DNQX) and miniature IPSCs recorded at --80 mV. mIPSCs were recorded in the presence of TTX.<br>The sound stimulation protocol was described in Cunha <i>et al., </i><i>Neuroscience</i>. 310:114-27. 2015.

本数据集包含假手术组与声音刺激组动物海马Schaffer-CA1突触处N-甲基-D-天冬氨酸受体（NMDA receptor）及AMPA/红藻氨酸（AMPA/kainate）介导电流的电流-电压（I-V）关系数据。
包含于-80 mV电位下记录的、取自假手术组与声音刺激组大鼠海马锥体神经元Schaffer-CA1突触的AMPA/kainate型兴奋性突触后电流（EPSCs）数据。
包含于70 mV电位下记录的、取自假手术组与声音刺激组大鼠海马锥体神经元Schaffer-CA1突触的NMDA型兴奋性突触后电流（EPSCs）数据；所有谷氨酸能电流均在印防己毒素（原文疑似笔误将picrotoxin写为picrotixi）存在条件下采集。
包含于-80 mV电位下记录的γ-氨基丁酸能（GABAergic）自发性抑制性突触后电流（记录时添加了DNQX）与微小抑制性突触后电流（mIPSCs）数据；其中微小抑制性突触后电流的采集额外添加了河豚毒素（TTX）。
本声音刺激方案已在Cunha等人发表于《Neuroscience》2015年第310卷第114-127页的研究中详细阐述。