Additional file 1 of A novel tRNA-derived fragment AS-tDR-007333 promotes the malignancy of NSCLC via the HSPB1/MED29 and ELK4/MED29 axes

Additional file 1: Table S1. Characteristics of NSCLC patients recruited for tRF and tiRNA sequencing. Table S2. Sequences of primers, inhibitor, and probes used in this study. Table S3. Expression levels of cytoplasmic AS-tDR-007333 between NSCLC tumor and adjacent tissues. Table S4. Expression levels of nucleus AS-tDR-007333 between NSCLC tumor and adjacent tissues. Table S5. Cox regression analysis on the association of AS-tDR-007333 with NSCLC prognosis. Table S6. Genes significantly regulated by AS-tDR-007333 over expression. Table S7. Gene ontology enrichment analysis of up-regulated genes by AS-tDR-007333. Table S8. Gene set enrichment analysis in AS-tDR-007333-overexpression cells vs. control cells.

补充文件1：表S1：入组开展tRF与tiRNA测序的非小细胞肺癌（NSCLC）患者的特征。
表S2：本研究所用引物、抑制剂及探针的序列。
表S3：细胞质AS-tDR-007333在非小细胞肺癌（NSCLC）肿瘤组织与癌旁组织中的表达水平。
表S4：细胞核AS-tDR-007333在非小细胞肺癌（NSCLC）肿瘤组织与癌旁组织中的表达水平。
表S5：AS-tDR-007333与非小细胞肺癌（NSCLC）预后相关性的Cox回归分析。
表S6：受AS-tDR-007333过表达显著调控的基因。
表S7：AS-tDR-007333上调基因的基因本体富集分析。
表S8：AS-tDR-007333过表达细胞与对照细胞的基因集富集分析。