alg-1 mutations cause retarded development.

aPresence and quality of cuticular alae structures were assayed by Normarski DIC optics. Only one side of each animal was scored.bAll animals contain maIs105 transgene which expresses an adult specific, col-19::GFP reporter.cThe quality of alae structure in lin-31; alg-1(anti) animals is extremely poor.dAverage number of seam cells per side was counted from pharynx to anus and therefore may contain 1 seam cell of the H lineage.elin-31(n1053) mutation suppresses the alg-1(anti) lethality by non-heterochronic mechanism.a. 采用诺曼斯基微分干涉差（Normarski DIC）光学显微镜检测角质侧嵴（cuticular alae）结构的存在状态与质量优劣，仅对每只受试线虫的单侧结构进行评分。 b. 所有受试线虫均携带maIs105转基因，该转基因可表达成年个体特异性的col-19::GFP报告基因。 c. lin-31; alg-1(anti)突变体线虫的侧嵴结构质量极差。 d. 统计范围为从咽至肛门区域的单侧表皮缝合细胞平均数量，因此统计结果可能包含1个H谱系来源的表皮缝合细胞。 e. lin-31(n1053)突变通过非异时性机制抑制alg-1(anti)的致死表型。