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资源简介：
p53 In Inflamatory Stress Response
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创建时间：
2005-11-30
相关数据集
GINS4 suppresses ferroptosis by antagonizing p53 acetylation with Snail
GINS4 suppresses ferroptosis by antagonizing p53 acetylation with Snail
integrated proteome resources80
GD3 Synthase Is a Master Regulator of Wild-Type p53âMediated Apoptosis and Mutant p53âMediated Tumorigenesis
We noted that the increased expression of GD3S enables breast cancer cells to evade cell death induced by the stabilization of p53 through Nutlin3A treatment, and we aim to explore the molecular mecha
NIAID Data Ecosystem20
p53 activates the long noncoding RNA Pvt1b to inhibit Myc and suppress tumorigenesis
P53 activation in response to cellular stress has long been known to result in downregulation of c-myc, a gene that is frequently overexpressed in cancer due to its role in promoting cellular prolifer
NIAID Data Ecosystem10
Differential expression of miRNAs derived from small RNA sequencing in HepG2 cell after activation of p53.
Differential expression of miRNAs derived from small RNA sequencing in HepG2 cell after activation of p53.
NIAID Data Ecosystem40
Synergistic activation of p53 by inhibition of MDM2 expression and DNA damage
The MDM2 oncogene encodes an inhibitor of the p53 tumor suppressor protein that regulates p53 in a negative feedback loop. MDM2 gene amplification and overexpression occur in several types of tumors a
PubMed Central1998-01-06 更新20
