Fragile X syndrome (FXS) is caused by transcriptional silencing of the FMR1 gene during embryonic development with the consequent loss of the encoded fragile X mental retardation protein (FMRP). The p
To elucidate birth-associated gene expression changes in the V-SVZ cells, scRNA-seq of the whole V-SVZ of the lateral wall of the lateral ventricles at E18.5, full-term P2, preterm P0, and preterm P3
Our work aims to characterize the role of Zrf1 in the generation and maintenance of neural progenitor cells (NPCs) Overall design: Gene expression profile of shCtrl and shZrf1 cells during generation
