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The Molecular Signature Induced by HMGA1 During Lymphoid Tumorigenesis

ChEBI2013-01-30 更新2026-07-21 收录

淋巴瘤发生机制

HMGA1分子特征

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-22922 数据链接链接失效反馈

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资源简介：

The Molecular Signature Induced by HMGA1 During Lymphoid Tumorigenesis

应用场景：

创建时间：

2013-01-30

淋巴瘤发生机制

转录因子调控网络

Many lymphoid malignancies arise from deregulated c-MYC expression in cooperation with additional genetic lesions. While many of these cooperative genetic lesions have been discovered and their functi

NIAID Data Ecosystem100

Pre-neoplastic somatic mutations including MYD88L265P in lymphoplasmacytic lymphoma [scRNA-seq]

淋巴瘤发生机制

肿瘤前体突变

Normal-cell counterparts of solid and myeloid tumors accumulate mutations years before disease onset; whether this occurs in B-lymphocytes prior to lymphoma remains uncertain. We sequenced multiple st

NIAID Data Ecosystem70

Actionable loss of SLF2 drives B-cell lymphomagenesis and impairs the DNA damage Response

淋巴瘤发生机制

SUMO化修饰

The DNA damage response (DDR) acts as a barrier to malignant transformation and is often impaired during tumorigenesis. Exploiting the impaired DDR can be a promising therapeutic strategy; however, th

NIAID Data Ecosystem50

Unrestricted SUMOylation drives lymphomagenesis

SUMOylation修饰

淋巴瘤发生机制

Unrestricted SUMOylation drives lymphomagenesis Overall design: Unrestricted SUMOylation drives lymphomagenesis

NIAID Data Ecosystem90

Gene expression from mouse models of B cell lymphomagenesis driven by gp130 signaling

淋巴瘤发生机制

基因表达调控

This study examines the transcriptional changes invoked by activation of gp130 signaling in different mouse models of B cell lymphomagenesis. In order to study the in vivo effects of aberrant activity

NIAID Data Ecosystem50

The Molecular Signature Induced by HMGA1 During Lymphoid Tumorigenesis

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