KDM5B ChIP-Seq project
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下载链接：
https://www.ncbi.nlm.nih.gov/sra/SRP051113
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资源简介：
Identify binding sites and epigenetic regulation of KDM5B histone demethylase
应用场景：
创建时间：
2017-09-17
相关数据集
KDM5B ChIP-Seq project. Mus musculus
Identify binding sites and epigenetic regulation of KDM5B histone demethylase
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The KDM5B histone H3 lysine 4 (H3K4) demethylase has been implicated in therapy resistance in multiple cancer types including breast cancer, but the underlying mechanism is poorly defined. Here we sho
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Extended self-renewal and accelerated reprogramming in the absence of Kdm5b [ChIP-Seq]
ES cell pluripotency is thought to be regulated in part by H3K4 methylation. However, it is unclear how H3K4 demethylation contributes to ES cell function and participates in iPS cell reprogramming. H
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KDM5B links cellular transcriptome heterogeneity to therapy resistance (ChIP-Seq). KDM5B links cellular transcriptome heterogeneity to therapy resistance (ChIP-Seq)
The KDM5B histone H3 lysine 4 (H3K4) demethylase has been implicated in therapy resistance in multiple cancer types including breast cancer, but the underlying mechanism is poorly defined. Here we sho
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KDM5B links cellular transcriptome heterogeneity to therapy resistance (ChIP-Seq)
The KDM5B histone H3 lysine 4 (H3K4) demethylase has been implicated in therapy resistance in multiple cancer types including breast cancer, but the underlying mechanism is poorly defined. Here we sho
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