<i>Mtb</i> Δ<i>fcr</i> mutants are severely impaired in developing phenotypic antibiotic tolerance under dormancy-inducing MS conditions.
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Mtb-WT, its fcr gene-knockout mutants (Δfcr1 & Δfcr2) and their complemented strains (C-fcr1 & C-fcr2) were subjected to MS for up to 18 days and antibiotic tolerance of the cultures against rifampicin (5 µg/ml) and isoniazid (0.8 µg/ml) were measured as previously described [11]. Average (±SD) from three different experiments (n = 3) is shown. MS, multiple-stress.
创建时间：
2012-12-14
