Induction and molecular signature of pathogenic Th17 cells

ChEBI2012-09-01 更新2026-07-21 收录

自身免疫病机制

Th17细胞

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-39820 数据链接链接失效反馈

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资源简介：

Induction and molecular signature of pathogenic Th17 cells

创建时间：

2012-09-01

total RNA-seq from T-helper 17 cell (ENCSR831GLL)

Th17细胞

单细胞RNA测序

h.CD4.Th17_DONOR 6: 0 hour no activation For data usage terms and conditions, please refer to http://www.genome.gov/27528022 and http://www.genome.gov/Pages/Research/ENCODE/ENCODE_Data_Use_Policy_for

NIAID Data Ecosystem90

Prmt5 deficiency inhibits CD4+T cells migration through Klf2-S1pr1 and ameliorates EAE disease (scATAC-seq)

自身免疫病机制

细胞迁移

Protein arginine methyltransferase 5 (Prmt5) is the major type II methyltransferase catalyzing symmetric dimethylation, regulates cell development, homeostatic and disease processes. Prmt5 inhibition

NIAID Data Ecosystem40

RNA sequencing in TH17 cells and modified TH17 cells

Th17细胞

RNA测序分析

To know how different genes are expressed in the TH17 cells treated by our functional molecule

NIAID Data Ecosystem10

16s rRNA of gut microbiome in PBC and pSS

微生物组

自身免疫病机制

This study aimed to analyze gut microbiome similarities and differences between primary biliary cholangitis (PBC) and primary Sjögren’s syndrome (pSS), exploring potential associations with disease pa

DataCite Commons2025-01-17 更新50

Reversing SKI–SMAD4-mediated suppression is essential for TH17 cell differentiation [ChIP-seq]. Mus musculus

Th17细胞

ChIP-seq分析

Disruption of Ski/Smad4 license Th17 differentiation. TGF-b-Ski-Smad4-RORgt-Th17 axis Overall design: Examination of Smad4 ChIP-seq in two different cells (WT vs.Smad4 KO) under IL6+TGFbR inhibitor co

NIAID Data Ecosystem20

Induction and molecular signature of pathogenic Th17 cells

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