Gene expression profiles in TICAM1 knockout and wild-type MEFs

ChEBI2016-07-29 更新2026-07-12 收录

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Gene expression profiles in TICAM1 knockout and wild-type MEFs

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2016-07-29

Gene expression profiles in TICAM1 knockout and wild-type MEFs

TICAM1 knockout and wild-type (TICAM1 knockout MEFs with restored TICAM1 expression) MEFs were treated by c-di-GMP or DMSO for 4 hours. Total RNA was analyzed via Illumina Mouse Ref-8 V2. Changes in g

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Expression profiling in Wild-type and C-JUN knockout MEFs treated with IFNgamma and cycloheximide

STAT1 is the major transcription factor (TF) driving response to exposure to IFNg. C-JUN is a TF which has been shown to bind with STAT1 and act as a partner TF. We have expression profiled WT and CJU

NIAID Data Ecosystem60

TICAM1 deficiency - HSE

Inborn errors of interferon immunity due to defects in toll like receptor 3 (TLR3)-mediated signaling underlie pathogenesis of herpes simplex virus type 1 (HSV1) encephalitis (HSE) in some children (N

reactome.org30

Gene expression profiles in TICAM1 knockout and wild-type MEFs. Mus musculus

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Gene transcription in wild-type (WT) and c-abl/arg knockout MEFs. Gene transcription in wild-type (WT) and c-abl/arg knockout MEFs

Abl kinases, which are required for multiple cellular processes, including proliferation, apoptosis, adhesion, cell migration, and stress responses, widely participate in the regulation of gene transc

NIAID Data Ecosystem30