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MiRNA profile of extracellular vesicles of senescent mesenchymal stromal cells.. Homo sapiens

NIAID Data Ecosystem2026-05-02 收录

细胞衰老机制

细胞外囊泡miRNA

数据链接：

https://www.ncbi.nlm.nih.gov/bioproject/PRJNA1117775 数据链接链接失效反馈

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资源简介：

In this study we analyzed changes in the expression of miRNAs in extracellular vesicles of early and late passage human mesenchymal stromal cells derived from chorionic tissue.

应用场景：

创建时间：

2024-05-29

细胞衰老机制

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Additional file 2 of miR-18a-5p derived from mesenchymal stem cells-extracellular vesicles inhibits ovarian cancer cell proliferation, migration, invasion, and chemotherapy resistance

卵巢癌分子机制

细胞外囊泡miRNA

Additional file 2: Table S2. miRNAs in the intersection of microarray GSE161784 and EVmiRNA database were identified.

Figshare2022-06-07 更新20

Senescence and Inflamm-Aging Are Associated With Endothelial Dysfunction in Men But Not Women With Atherosclerosis. Senescence and Inflamm-Aging Are Associated With Endothelial Dysfunction in Men But Not Women With Atherosclerosis

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细胞衰老机制

Coronary artery disease (CAD) is a major cause of death worldwide, and more prevalent in men than in women. This study examines explored the differences in endothelium-dependent relaxation and associa

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Small extracellular vesicles are key regulators of non-cell autonomous intercellular communication in senescence via the interferon protein, IFITM3

细胞衰老机制

细胞外囊泡通讯

Senescence is a cellular phenotype present in health and disease, characterized by a stable cell cycle arrest and an inflammatory response, denominated senescence-associated secretory phenotype (SASP)

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Molecular chaperone Hsp90 supports adaptive cell size increase in response to chronic stress

慢性应激适应

细胞衰老机制

Hsf1, well known for its role in acute stress responses, is required for the cell size increase, and that the molecular chaperone Hsp90 is essential for coupling the cell size increase to augmented tr

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MiRNA profile of extracellular vesicles of senescent mesenchymal stromal cells.. Homo sapiens

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