HMGA1 Chromatin Regulators Drive MPN Progression
收藏NIAID Data Ecosystem2026-05-01 收录
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This SuperSeries is composed of the SubSeries listed below. Refer to individual Series
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2024-01-08
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Additional file 1 of Associations of the circulating levels of cytokines with the risk of myeloproliferative neoplasms: a bidirectional mendelian-randomization study
Supplementary Material 1.
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Evolution of the cancer epigenome in myeloproliferative neoplasms 2
Whole genome sequencing and bisulphite sequencing of haematopoietic coloniesEGA study EGAS00001002372
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ZRSR2-loss causes aberrant splicing in a JAK2V617F-driven myeloproliferative neoplasm but is not sufficient to drive disease progression. ZRSR2-loss causes aberrant splicing in a JAK2V617F-driven myeloproliferative neoplasm but is not sufficient to drive disease progression
This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series
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Jak2V617F Reversible Activation Shows an Essential Requirement for Jak2V617F in Myeloproliferative Neoplasms. Jak2V617F Reversible Activation Shows an Essential Requirement for Jak2V617F in Myeloproliferative Neoplasms
Janus kinases (JAKs) mediate cytokine signaling, cell growth and hematopoietic differentiation. Gain-of-function mutations activating JAK2 signaling are seen in the majority of myeloproliferative neop
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Fibroblast growth factor receptor 1 is fused to FIM in stem-cell myeloproliferative disorder with t(8;13)(p12;q12)
Chromosome 8p11–12 is the site of a recurrent breakpoint in a myeloproliferative disorder that involves lymphoid (T- or B-cell), myeloid hyperplasia and eosinophilia, and evolves toward acute leukemia
PubMed Central1998-05-12 更新40
