Aims: Insulin resistance, dyslipidemia and hypertension are independent mediators of endothelial dysfunction. It is incompletely defined whether dyslipidemia and hypertension in addition
To investigate the potential molecular mechanism of diabetic endothelial dysfunction, RNA-Sequencing Analysis was used to profile influence of high glucose and MSC to huvec
This study aimed to elucidate the mechanisms underlying the anti-diabetic effects of Mathurameha against high glucose-induced endothelial dysfunction by SWATH-MS. The EA.hy926 cells were treated with
The aim of our study is to explore the involvement of PPARα and PPARγ in Ang II-induced endothelial injury. We found that Ang II significantly elevated the oxidative stress in HUVECs, causing apoptosi
Data are presented as mean with 95% Confidence interval (CI). n: number of participants fulfilling the specific criteria; No MetSyn: participants not fulfilling the specific criteriafor metabolic synd