Gene Expression in Castration-Resistant C4-2 Cells after AR Knockdown

ChEBI2010-06-23 更新2026-07-21 收录

耐药性前列腺癌

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数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-13332 数据链接链接失效反馈

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资源简介：

Gene Expression in Castration-Resistant C4-2 Cells after AR Knockdown

创建时间：

2010-06-23

GREB1 amplifies androgen receptor output in prostate cancer and contributes to antiandrogen resistance. GREB1 amplifies androgen receptor output in prostate cancer and contributes to antiandrogen resistance

耐药性前列腺癌

雄激素受体信号

Genomic amplification of the androgen receptor (AR) is an established mechanism of antiandrogen resistance in prostate cancer. Here we show that the magnitude of AR signaling output, independent of AR

NIAID Data Ecosystem70

Time-course effect of estradiol and ERa17p on Early Gene expression in SKBR3 cells

乳腺细胞分子机制

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Time-course effect of estradiol and ERa17p on Early Gene expression in SKBR3 cells

ChEBI2012-07-30 更新20

Phosphorylated and SUMO-deficient progesterone receptors drive a gene expression profile important for breast cancer progression

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乳腺癌分子机制

Phosphorylated and SUMO-deficient progesterone receptors drive a gene expression profile important for breast cancer progression

ChEBI2012-06-06 更新00

Drug-induced epigenomic plasticity reprograms circadian rhythm regulation to drive prostate cancer towards androgen-independence [Cell line ChIP-Seq]

耐药性前列腺癌

昼夜节律表观遗传调控

In prostate cancer, androgen receptor (AR)-targeting agents are very effective in various stages of the disease. However, therapy resistance inevitably occurs and little is known about how tumor cells

NIAID Data Ecosystem70

Convergence of oncogenic and hormone receptor pathways promotes pro-metastatic phenotypes.

癌症转移机制

激素受体信号通路

Convergence of oncogenic and hormone receptor pathways promotes pro-metastatic phenotypes.

ChEBI2013-03-31 更新00