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IFN-g and Androgens Synergistically Disrupt Mitochondrial Function in Murine Myocytes

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We report here, aberrant cardiovascular activity in vivo in an autoimmune mouse model with persistent low-level expression of IFN-g (designated ARE mice) linked with androgen exposure, results in a mitochondrial and metabolic deficiency. 4 WT and 4 ARE-/- male mice hearts are examined via bulk RNAseqencing

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