Loss-of-function mutations of p16(INK4a) have been identified in a large number of human tumors. An established biochemical function of p16 is its ability to specifically inhibit cyclin D-dependent ki
This study was designed to look for the RNAs that directly bind with QKI HA tagged QKI-5 and QKI-6 were overexpressed in QPP GSCs and were applied to PAR-CLIP protocol. RNAs were exacted and sent for