White-to-brown metabolic conversion of human adipocytes by JAK inhibition

ChEBI2014-10-31 更新2026-07-21 收录

脂肪细胞代谢

JAK抑制代谢调控

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-57896 数据链接链接失效反馈

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资源简介：

White-to-brown metabolic conversion of human adipocytes by JAK inhibition

创建时间：

2014-10-31

Low n6/n3 Ratio Consumption of Neonates Establishes Lipid Oxidative Metabolism in Adipocyte Precursor Cells [single-cell RNA-seq]

脂肪细胞代谢

单细胞转录组学

Adipocyte precursor cells from 12-day-old pups were characterized by single cell RNA sequencing Subcutaneous adipose tissue was enzymatically digested producing a stromal vasular pellet that was flow

NIAID Data Ecosystem70

Dysfunctional Adipocytes Promote Tumor Progression Through YAP/TAZ-dependent Cancer-Associated Adipocyte Transformation

肿瘤微环境

脂肪细胞代谢

Unedited raw data of [NCOMMS-23-27303B]

Figshare2024-05-15 更新30

Adipocyte-Specific Ablation Of PU.1 Promotes Energy Expenditure and Ameliorates Metabolic Syndrome In Aging Mice

脂肪细胞代谢

代谢综合征小鼠模型

Here, we investigated the role of adipocyte PU.1 in the development of age-associated metabolic syndrome. We generated mice with adipocyte specific PU.1 knockout, assessed metabolic changes in young a

Alliance of Genome Resources10

Real-time quantitative PCR analysis of 3T3-L1 cells treated with Interleukin-15 (IL-15)

脂肪细胞代谢

细胞因子基因表达分析

Differentiated 3T3-L1 adipocytes were either treated 0ng/ml or 500ng/ml rmIL-15 for 24 hours (n=3/group) qPCR gene expression profilling performed in vitro using recombinant murine (rm)IL-15 treated (

NIAID Data Ecosystem50

Insulin-stimulated glucose uptake in recombinant MMP12 treated 3T3-L1 cells. Insulin-stimulated glucose uptake in recombinant MMP12 treated 3T3-L1 cells

胰岛素抵抗机制

脂肪细胞代谢

Microbiota contributes to induction of type 2 diabetes by high-fat/high-sugar (HFHS), but which organs/pathways are impacted by microbiota remains unknown. Using multi-organ network and transkingdom a

NIAID Data Ecosystem30

White-to-brown metabolic conversion of human adipocytes by JAK inhibition

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