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Identification of microRNAs dysregulated in cellular senescence driven by endogenous genotoxic stress

ChEBI2013-06-29 更新2026-07-21 收录

microRNA表达谱分析

细胞衰老机制

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-48417 数据链接链接失效反馈

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资源简介：

Identification of microRNAs dysregulated in cellular senescence driven by endogenous genotoxic stress

应用场景：

创建时间：

2013-06-29

细胞衰老机制

STAT信号通路

SOCS1 plays a role in cellular senescence. Knocking down SOCS1 in senescence induced by the STAT5 oncogene results in senescence bypass by preventing p53 activation IMR90 cells were infected with retr

NIAID Data Ecosystem80

MicroRNA map of Paralichthys olivaceus spleen at 6h and 24h after Streptococcus iniae infection

鱼类免疫应答

microRNA表达谱分析

In this study, we applied high-throughput sequencing technology to examine miRNAs in Japanese flounder (Paralichthys olivaceus) infected with Streptococcus iniae at different times. Overall design: Th

NIAID Data Ecosystem50

Human Keratinocytes That Express hTERT and Also Bypass a p16(INK4a)-Enforced Mechanism That Limits Life Span Become Immortal yet Retain Normal Growth and Differentiation Characteristics

角质形成细胞永生化

细胞衰老机制

Normal human cells exhibit a limited replicative life span in culture, eventually arresting growth by a process termed senescence. Progressive telomere shortening appears to trigger senescence in norm

PubMed Central40

Age-associated dysregulation of peroxisomal matrix protein importing impairs intestinal stem cell differentiation and gut functions

细胞衰老机制

肠道干细胞

Age-associated dysregulation of peroxisomal matrix protein importing impairs intestinal stem cell differentiation and gut functions

integrated proteome resources50

ATF3 drives senescence by reconstructing accessible chromatin profiles

细胞衰老机制

染色质可及性分析

We depicted the landscapes of both chromatin accessibility and gene expression to reveal gene regulatory networks in human umbilical vein endothelial cell (HUVEC) senescence and found that chromatin a

NIAID Data Ecosystem40

Identification of microRNAs dysregulated in cellular senescence driven by endogenous genotoxic stress

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