Emodin inhibits macrophage classical activation [Control-LPS-IFNg-Emodin]

ChEBI2016-07-04 更新2026-07-12 收录

免疫药理学

巨噬细胞调控

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-73310 数据链接链接失效反馈

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资源简介：

Emodin inhibits macrophage classical activation [Control-LPS-IFNg-Emodin]

创建时间：

2016-07-04

Human Amniotic Epithelial Cell Transplantation Induces Markers of Alternative Macrophage Activation and Reduces Established Hepatic Fibrosis

肝纤维化细胞治疗

巨噬细胞调控

Chronic hepatic inflammation from multiple etiologies leads to a fibrogenic response that can progress to cirrhosis and liver failure. Transplantation of human amniotic epithelial cells (hAEC) from te

NIAID Data Ecosystem60

Gene expression in zebrafish as a result of Lamotrigin (epilepsy drug) exposure

免疫药理学

鱼类基因表达分析

The antiepileptic medication Lamotrigin is known to increase immunoglobulins in humans. The aim of this current study is to to examine if zebrafish larvae exposed to Lamotrigin show immunological effe

NIAID Data Ecosystem60

CD40 induces PIR-A+ macrophages to promote chronic rejection of cardiac transplantation

移植免疫

巨噬细胞调控

To investigate the mechanism PIR-A+ macrophages regulated by CD40 in chronic rejection of cardiac transplantation, we performed gene expression profiling analysis using data obtained from RNA-seq of b

NIAID Data Ecosystem10

SMS TMT surfaceome comparison of ex vivo derived M1 macrophages treated with dexamethasone

免疫蛋白质组学

巨噬细胞调控

Triplicate samples of donor matched ex vivo generated macrophage subtype M1 cells treated with and without dexamethasone compared against the M0 control analysed using TMT-based 11plex quantitative pr

NIAID Data Ecosystem60

regulatory changes induced by miR-223 in a monocyte/macrophage cell line.

巨噬细胞调控

RNA靶向治疗

We used a multi-omics approach combining transcriptomics, proteomics and metabolomics to study the impact of over-expression and inhibition of the microRNA miR-223, a pleiotropic regulator of metaboli

NIAID Data Ecosystem30

Emodin inhibits macrophage classical activation [Control-LPS-IFNg-Emodin]

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