Abnormal DNA levels, such as aneuploidy and polyploidy, can indicate cellular transformation and cancer; however, the mechanisms remain poorly understood. All tumor viruses inherently cause abnormal D

NIAID Data Ecosystem20

Host range restrictions of oncogenes: myc genes transform avian but not mammalian cells and mht/raf genes transform mammalian but not avian cells.

肿瘤病毒学

细胞转化实验

The host range of retroviral oncogenes is naturally limited by the host range of the retroviral vector. The question of whether the transforming host range of retroviral oncogenes is also restricted b

PubMed Central1996-07-23 更新10

SERBP1 deacetylation inhibits ferroptosis to promote KSHV-induced cellular transformation by decaying lipoyltransferase 2 mRNA

肿瘤病毒学

铁死亡调控

Ferroptosis, a defensive strategy employed by the host to restrict pathogenic infections, has been implicated in the development and therapeutic responses of various types of tumors. However, the role

NIAID Data Ecosystem20

Activation of glucocorticoid receptor signaling inhibits KSHV-induced inflammation and tumorigenesis

肿瘤病毒学

炎症信号通路靶向治疗

The hallmark of Kaposi’s sarcoma (KS), the most common cancer in HIV-infected patients caused by Kaposi’s sarcoma-associated herpesvirus (KSHV) infection, is hyperinflammation. However, the role of in

NIAID Data Ecosystem10

KSHV-infected invasive bladder cancer cell: Control vs. KSHV-infected

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