Dysregulation of alternative splicing in prostate cancer is linked to transcriptional programs activated by canonical oncogenic transcription factors. However, the relative contribution of these facto
Additional file 4: Table S4. rMATS results for PRJEB2449 cohort. (a) A summary of the total number of events, significant annotated splicing events, and their associated parent genes as identified usi
HTA2.0 affy array was used to study the effect of androgen inhibitors on alternative splicing in LNCaP cells Overall design: To study the effect of pharmacological manipulation of AR signaling in pros
We sought to define the landscape of alternative pre-mRNA splicing in prostate cancers and the relationship of exon choice to known genetic driver alterations. To do so, we compiled a meta-dataset com