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ALKBH5 SUMOylation-mediated FBXW7 m6A modification regulates epithelial cells senescence during 1-nitropyrene-induced pulmonary fibrosis

NIAID Data Ecosystem2026-05-01 收录

纤维化分子机制

m6A表观遗传学

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE251885 数据链接链接失效反馈

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This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

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创建时间：

2024-03-31

白血病耐药机制

m6A表观遗传学

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Renal fibrosis is a widely used pathological indicator of progressive chronic kidney disease (CKD), and renal fibrosis mediates most progressive renal diseases as a final pathway. Nevertheless, the ke

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Additional file 14: Table S7. of A pilot systematic genomic comparison of recurrence risks of hepatitis B virus-associated hepatocellular carcinoma with low- and high-degree liver fibrosis

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HBV integration sites identified in BGI, TCGA, and ICGC cohorts. (XLSX 92 kb)

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Chitinase-3 Like 1 Is a Profibrogenic Factor Overexpressed in Aging Livers and in Patients with Cirrhosis in the Aging Liver and in Patients with Liver Cirrhosis

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Older age at the time of infection with hepatitis viruses is associated with an increased risk of liver fibrosis progression, but the mechanisms remain unclear. We hypothesized that the pace of liver

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Next Generation Sequencing of Lung Primary Fibroblast Responding to Eosinophil-Degranulation Products

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Methods: Two human lung primary fibroblast (HLF) lines were activated for 24 h with eosinophil conditioned media obtained from two blood eosinophil donors. Total RNA was extracted from HLF and treated

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ALKBH5 SUMOylation-mediated FBXW7 m6A modification regulates epithelial cells senescence during 1-nitropyrene-induced pulmonary fibrosis

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