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Influenza infection rewires energy metabolism and induces browning features in adipose cells and tissues

ChEBI2019-07-20 更新2026-07-12 收录

感染代谢学

棕色脂肪细胞

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-6646 数据链接链接失效反馈

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Influenza infection rewires energy metabolism and induces browning features in adipose cells and tissues

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创建时间：

2019-07-20

棕色脂肪细胞

转录因子调控

The transcription factor nuclear factor I-A (NFIA) is a regulator of brown adipocyte differentiation. Here we show that the most C-terminal 17 amino acid residues of NFIA (which we named pro#3 domain)

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Cell-autonomous light sensitivity via Opsin3 regulates fuel utilization in brown adipocytes

棕色脂肪细胞

能量代谢

Opsin3 (Opn3) is a transmembrane heptahelical G-protein-coupled receptor (GPCR) with the potential to produce a non-visual photoreceptive effect. Interestingly, anatomical profiling of GPCRs reveals

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NFIA determines the cis-effect of genetic variation on adipocyte browning in mice [ChIP-Seq]

棕色脂肪细胞

遗传调控

Thermogenic brown and beige adipocytes counteract obesity by enhancing energy dissipation via uncoupling proein-1 (Ucp1). However, the effect of genetic variation on these cells, a major source of dis

NIAID Data Ecosystem20

Unravelling the developmental roadmap towards human brown adipose tissue

棕色脂肪细胞

干细胞分化

We have developed and characterised functionally and molecularly a new chemically-defined protocol for the differentiation of human pluripotent stem cells (hPSCs) into brown adipocytes (BAs) that over

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Cistromic and epigenomic changes in Prdm16 knock-out brown adipose tissue. Mus musculus

表观遗传调控

棕色脂肪细胞

Prdm16 is a transcription factor that drives a complete program of brown adipocyte differentiation, but the mechanism by which Prdm16 activates gene transcription remains unknown. Utilizing ChIP-seq t

NIAID Data Ecosystem20

Influenza infection rewires energy metabolism and induces browning features in adipose cells and tissues

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