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Yeast response to the DNA damaging beta-lapachone
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创建时间：
2011-12-31
相关数据集
Negative control of CSL gene transcription by stress/DNA damage response and p53 [ChIP-Seq]
CSL is a key transcription factor, mostly acting as a repressor. While known as main effector of Notch signaling, it can also play Notch-independent functions. Despite the wide interest in CSL, the me
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DNA-PKcs RNASeq data: DNA-PKcs wilde-type or kinase-dead protein regulate basal and etoposide-induced gene expression changes
Maintenance of the genome is essential for cell survival, and impairment of the DNA damage response is associated with multiple pathologies including cancer and neurological abnormalities. DNA-PKcs is
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Chk2 is dispensable for p53-mediated G(1) arrest but is required for a latent p53-mediated apoptotic response
In response to genotoxic stress, mammalian cells can activate cell cycle checkpoint pathways to arrest the cell for repair of DNA damage or induce apoptosis to eliminate damaged cells. The checkpoint
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Translational and transcriptional responses of fission yeast to MMS
Translational and transcriptional responses of fission yeast to MMS
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Genome-wide redistribution of H3K27me3 is linked to genotoxic stress and defective growth. Neurospora crassa
H3K9 methylation directs heterochromatin formation by recruiting multiple HP1-containing complexes that deacetylate histones and methylate cytosine bases in DNA. In Neurospora crassa, a single H3K9 me
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