Dual Pten/p53 suppression enhances Notch signaling and sarcoma progression

ChEBI2013-01-01 更新2026-07-21 收录

肿瘤分子机制

肿瘤病理研究

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-42103 数据链接链接失效反馈

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资源简介：

Dual Pten/p53 suppression enhances Notch signaling and sarcoma progression

创建时间：

2013-01-01

Transcriptome profile of human colorectal adenomas.

结直肠肿瘤转录组学

肿瘤分子机制

Background: Colorectal cancers are believed to arise predominantly from adenomas. Although these precancerous lesions have been subjected to extensive clinical, pathological, and molecular analyses, l

NIAID Data Ecosystem30

Microarray of Ba/F3-MPL overexpressing or not different CALR mutation

肿瘤分子机制

基因突变功能

Microarray of Ba/F3-MPL overexpressing or not different CALR mutation

ChEBI2020-07-01 更新00

Prognostic Value and Molecular Mechanisms of Proteasome 26S Subunit, Non-ATPase Family Genes for Pancreatic Ductal Adenocarcinoma Patients after Pancreaticoduodenectomy

胰腺癌预后生物标志物

肿瘤分子机制

Objective: Pancreatic cancer (PC) is an extremely malignant tumor with similar morbidity and mortality and lack of an effective treatment. This study explored the prognostic value and molecular

Taylor & Francis Group2024-02-07 更新00

Tumors with TSC mutations are sensitive to CDK7 inhibition through NRF2 and glutathione depletion. Tumors with TSC mutations are sensitive to CDK7 inhibition through NRF2 and glutathione depletion

肿瘤分子机制

敏感性分析

We report the global gene expression effects in TSC-deficient and TSC wild type cells after treatemnet with. CDK7 inhibitor(THZ1), we performed RNA-Seq on TSC1-null HCV.29 cells and TSC1-intact HCV.29

NIAID Data Ecosystem30

identification of TGCT genes involved in initiation and maintenance of transformed germ cells.

睾丸生殖细胞肿瘤基因

肿瘤分子机制

identification of TGCT genes involved in initiation and maintenance of transformed germ cells.

ChEBI2009-12-30 更新00