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Transcriptional profilling of hESC with and without 17q gain upon induction of replicative stress through HU treatment

NIAID Data Ecosystem2026-03-12 收录

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17q增益基因表达

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE150647 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

应用场景：

创建时间：

2021-08-11

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DNA损伤应答

DNA damage results in the activation of a transcriptional program that promotes DNA repair and stalls cell cycle progression. To determine which transcriptional programs are induced by replication str

NIAID Data Ecosystem30

3D genome organization, transcription and aberrant DNA replication contribute to genome instability at fragile sites. 3D genome organization, transcription and aberrant DNA replication contribute to genome instability at fragile sites

脆性位点机制

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Common fragile sites (CFSs) are regions susceptible to replication stress and are hotspots for chromosomal instability in cancer. Several features characterizing CFSs have been associated with their i

NIAID Data Ecosystem30

Mouse ES cell line transcriptome changes upon replication stress. Mus musculus

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胚胎干细胞转录组

To understand whether activation of ATR-dependent replication stress response could result in global transcriptional alterations similar to that of 2C-like cells, we performed whole transcriptional pr

NIAID Data Ecosystem30

Transcriptional profilling of hESC with and without 17q gain upon induction of replicative stress through HU treatment. Transcriptional profilling of hESC with and without 17q gain upon induction of replicative stress through HU treatment

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人类胚胎干细胞基因研究

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

NIAID Data Ecosystem20