This study provide an opportunity to elucidate the genetic control of fetal implantation and improve our understanding of fetal implantation and gestation maintenance, thus make further improvement fo
The Wnt1/C2mE transgenic mouse was used as a genetic model of spontaneous gastric carcinogenesis [15]. SLD5+/−/Wnt1/C2mE transgenic mice were generated by mating Wnt1/C2mE transgenic mice and SLD5+/−
Inositol 1,4,5‐trisphosphate receptors (IP3Rs) are a family of intracellular Ca2+ release channels located on the ER membrane, which in mammals consist of 3 different subtypes (IP3R1, IP3R2, and IP3R3