Replication data for The Role of Senescence in Experimental Periodontitis at the Causal Level: An in Vivo Study

DataONE2025-11-25 更新2025-12-06 收录

牙周炎研究

衰老机制

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supporting raw data for Western blot and immunofluorescence

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2025-11-28

线粒体应激长寿模型转录组数据

线粒体应激

衰老机制

线粒体作为细胞的能量工厂，为机体运转提供动力的同时也参与代谢调控和信号转导。在压力条件下，线粒体通过与细胞核之间的交流，诱导线粒体未折叠蛋白反应(mitochondrial unfolded protein response, UPRmt)，维持线粒体稳态平衡，从而促使细胞适应代谢压力。尽管线粒体严重受损对机体是有害的，但在线虫、果蝇及小鼠中都有研究表明轻微降低线粒体电子传递链(electron

中国科学院中国科学技术大学科学数据中心260

Additional file 1: of Assessing the progression of chronic periodontitis using subgingival pathogen levels: a 24-month prospective multicenter cohort study

牙周炎研究

伦理审查

List of the ethics committees and approval numbers. (XLSX 12 kb)

DataCite Commons2024-12-18 更新100

Coupling shRNA screens with single-cell RNA-Seq identifies mechanisms regulating senescence during reprogramming. Coupling shRNA screens with single-cell RNA-Seq identifies mechanisms regulating senescence during reprogramming

细胞重编程

衰老机制

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

NIAID Data Ecosystem70

NOTCH-mediated non-cell autonomous regulation of chromatin structure during senescence [ChIP-Seq]. Homo sapiens

染色质结构

衰老机制

We describe the accessible chormatin landscape in RAS-induced (RIS) and NOTCH induced senescence (NIS) using ATAC-seq. By expressing active NOTCH (N1ICD) in the context of RIS, we find that N1ICD anta

NIAID Data Ecosystem160

皮肤免疫

衰老机制

Dermal fibroblasts (dFB) resist infection by locally differentiating into adipocytes and producing the antimicrobial peptide cathelicidin in response to S. aureus. We found that neonatal dFB were high

NIAID Data Ecosystem100

Replication data for The Role of Senescence in Experimental Periodontitis at the Causal Level: An in Vivo Study

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