Study of MicroRNA-124 in Patients with Lupus Nephritis
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Study of MicroRNA-124 in Patients with Lupus Nephritis
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2023-01-24
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Loss of interleukin-1 beta is not protective in the lupus-prone NZM2328 mouse model. Loss of interleukin-1 beta is not protective in the lupus-prone NZM2328 mouse model
RNA-sequencing analysis identified upregulation of TNF and IL-17 signaling pathways specifically in females lacking IL-1β. Increases in these signaling pathways are also found in female patients with
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Supplementary Material for: Clinicopathological characteristics and outcomes of patients with crescentic lupus nephritis
Background：Crescentic lupus nephritis (cLN) is an uncommon and severe phenotype in LN with limited data on outcomes and relevant risk factors. Methods: Eighty-three cases of biopsy-proven LN with ≥50
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Progress in diagnosis and treatment of lupus nephritis complicated with thrombotic microangiopathy
Lupus nephritis (LN) is one of the common secondary glomerulonephritis in clinic. Thrombotic microangiopathy (TMA)is a clinicopathological syndrome with high mortality and involving multiple systems.
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Additional file 2 of Proteomic analysis in lupus mice identifies Coronin-1A as a potential biomarker for lupus nephritis
Additional file 2. Discovery proteomics data (accession number, protein names, p value, q-value, fold change status of identified proteins). List of common proteins between mouse kidney discovery prot
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Demographic data of the SLE patients included in the study at the time-point of blood sampling.
SLE-G1: Mild SLE with skin and musculoskeletal involvement, SLE-G2: More severe SLE with serositis, vasculitis, CNS but not kidney involvement, SLE-G3: SLE glomerulonephritis.
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