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Increased PTCHD4 expression via m6A modification of PTCHD4 mRNA promotes senescent-cell survival

NIAID Data Ecosystem2026-05-02 收录

衰老细胞存活机制

m6A表观转录组学

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE247621 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

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创建时间：

2024-12-18

m6A表观转录组学

卵巢癌耐药性

Additional file 2 The differentially enriched m6A-peaks of MeRIP-seq (|log2 fold change (FC)| ≥1, p<0.05).

NIAID Data Ecosystem40

Widespread N6-methyladenosine-dependent RNA Structural Switches Regulate RNA-Protein Interactions

m6A表观转录组学

RNA-蛋白质相互作用

We show that N6-methyladenosine (m6A), the most abundant internal modification in mRNA/lncRNA with still poorly characterized function, alters RNA structure to facilitate the access of RBM for heterog

NIAID Data Ecosystem30

Integrated analysis of the transcriptome wide m6A methylome in Peripheral nerve injury

周围神经损伤

m6A表观转录组学

N6-methyladenosine(m6A) is the most plentiful internal RNA modification, especially in eukaryotic messenger RNA (mRNA), which plays pivotal roles in mRNA life cycle and nerve development processes. In

Alliance of Genome Resources10

miCLIP analysis of m6A sites in LNCaP and RWPE cells

m6A表观转录组学

前列腺癌分子机制

Genomic and transcriptomic alterations are insufficient to explain the variance in protein expression seen in cancer. Recent evidence has highlighted the role of N6-methyladenosine (m6A) in the regula

NIAID Data Ecosystem30

Data_Sheet_7_N6-methyladenosine modulation classes and immune microenvironment regulation in ischemic stroke.XLSX

缺血性卒中免疫微环境

m6A表观转录组学

N6-methyladenosine (m6A) modifications play an important role in the differentiation and regulation of immune cells. However, research on m6A in ischemic stroke (IS) is still in its infancy, and their

NIAID Data Ecosystem10

Increased PTCHD4 expression via m6A modification of PTCHD4 mRNA promotes senescent-cell survival

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