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Genomic aberrations in myeloproliferative neoplasms
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创建时间：
2015-11-30
相关数据集
Additional file 1 of Associations of the circulating levels of cytokines with the risk of myeloproliferative neoplasms: a bidirectional mendelian-randomization study
Supplementary Material 1.
Figshare2024-08-15 更新80
Supplementary Table from LKB1/ STK11 Is a Tumor Suppressor in the Progression of Myeloproliferative Neoplasms
Supplementary Table from LKB1/STK11 Is a Tumor Suppressor in the Progression of Myeloproliferative Neoplasms
NIAID Data Ecosystem50
Fibroblast growth factor receptor 1 is fused to FIM in stem-cell myeloproliferative disorder with t(8;13)(p12;q12)
Chromosome 8p11–12 is the site of a recurrent breakpoint in a myeloproliferative disorder that involves lymphoid (T- or B-cell), myeloid hyperplasia and eosinophilia, and evolves toward acute leukemia
PubMed Central1998-05-12 更新40
Jak2V617F Reversible Activation Shows an Essential Requirement for Jak2V617F in Myeloproliferative Neoplasms. Jak2V617F Reversible Activation Shows an Essential Requirement for Jak2V617F in Myeloproliferative Neoplasms
Janus kinases (JAKs) mediate cytokine signaling, cell growth and hematopoietic differentiation. Gain-of-function mutations activating JAK2 signaling are seen in the majority of myeloproliferative neop
NIAID Data Ecosystem60
Evolution of the cancer epigenome in myeloproliferative neoplasms 2
Whole genome sequencing and bisulphite sequencing of haematopoietic coloniesEGA study EGAS00001002372
NIAID Data Ecosystem50
