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Epstein-Barr Virus Exploits Intrinsic B-Lymphocyte Transcription Programs to Achieve Immortal Cell Growth

ChEBI2011-07-01 更新2026-07-21 收录

肿瘤病毒学

B细胞淋巴瘤机制

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-29498 数据链接链接失效反馈

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Epstein-Barr Virus Exploits Intrinsic B-Lymphocyte Transcription Programs to Achieve Immortal Cell Growth

应用场景：

创建时间：

2011-07-01

B细胞淋巴瘤机制

基因表达谱分析

Inactivating mutations of the gene encoding for the CREBBP acetyltransferase are highly frequent in diffuse large B cell lymphoma (DLBCL, 30% of cases) and follicular lymphoma (FL, 60% of cases), the

NIAID Data Ecosystem40

Enhancer Sequence Variants and Transcription Factor Deregulation Synergize to Construct Pathogenic Regulatory Circuits in B Cell Lymphoma

B细胞淋巴瘤机制

增强子-转录因子调控

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

NIAID Data Ecosystem30

Host range restrictions of oncogenes: myc genes transform avian but not mammalian cells and mht/raf genes transform mammalian but not avian cells.

肿瘤病毒学

细胞转化实验

The host range of retroviral oncogenes is naturally limited by the host range of the retroviral vector. The question of whether the transforming host range of retroviral oncogenes is also restricted b

PubMed Central1996-07-23 更新10

Genome-wide Virus-integration Analysis Reveals A Common Oncogenic Insertional Mechanism of HPV, HBV and EBV. viral metagenome

病毒整合分析

肿瘤病毒学

Genome-wide Virus-integration Analysis Reveals A Common Oncogenic Insertional Mechanism of HPV, HBV and EBV

NIAID Data Ecosystem40

SERBP1 deacetylation inhibits ferroptosis to promote KSHV-induced cellular transformation by decaying lipoyltransferase 2 mRNA

肿瘤病毒学

铁死亡调控

Ferroptosis, a defensive strategy employed by the host to restrict pathogenic infections, has been implicated in the development and therapeutic responses of various types of tumors. However, the role

NIAID Data Ecosystem20

Epstein-Barr Virus Exploits Intrinsic B-Lymphocyte Transcription Programs to Achieve Immortal Cell Growth

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