Additional file 13 of Knockout of thyroid hormone receptor alpha a (thraa) enhances cardiac regeneration in zebrafish through metabolic and hypoxic regulation
Single cell RNA seq of uninjured and injured (wnt-inhibited and non-inhibited) zebrafish hearts accompanied by CRISPR-based lineage tracing obtained by Hu, Lelek, Spanjaard et al. (Nature Genetics 202
longfin mutant zebrafish grow and regenerate abnormally long fins. We sought to determine misregulated transcripts in the distal regenerate of 96 hpa caudal fins that could contribute to fin overgrowt
Background: Zebrafish can faithfully regenerate injured fins through the formation of a blastema, a mass of proliferative cells that can grow and develop into the lost body part. After amputation, var
We compare the brain and fin RNA-seq profiles of wild-type and scn8ab mutant zebrafish, which exhibit locomotion and fin regeneration defects. Overall design: Biological duplicate brain and fin profil
We report increased expression of mitochondrial biogenesis genes in regnerating larval fin fold Overall design: Larval fin fold mRNA profiles of 3-day old control and fin amputated zebrafish