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Simulation runs for different neural disruption types.Speeds are provided in [m/s].
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2021-08-10
相关数据集
Mechanistic differences in neuropathic pain modalities revealed by correlating behavior with global expression profiling [microarray]
Chronic neuropathic pain is a major morbidity of neural injury, yet its mechanisms are incompletely understood. Hypersensitivity to previously non-noxious stimuli (allodynia) is a common symptom. Here
NIAID Data Ecosystem110
Additional file 3 of Positive interaction between GPER and β-alanine in the dorsal root ganglion uncovers potential mechanisms: mediating continuous neuronal sensitization and neuroinflammation responses in neuropathic pain
Additional file 3: Table S2. Primers Information of qPCR Experiment.
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Data and Code; Activity-dependent node of Ranvier shortening and myelin increases enhance thalamocortical synchrony in motor learning
This deposit contains the computational model (available at https://github.com/AttwellLab/MyelinatedAxonModel) used for the axonal conduction simulations reported in this paper, together with the outp
Zenodo2026-05-08 更新30
Transcriptomic profiling in mice with CB1 deletion in primary sensory neurons
in this study, we investigated whether CB1R conditional knockout from peripheral sensory neurons (CB1cKO) in mice may present mechanical allodynia, thermal hyperalgesia and change transcriptome expres
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The role of m6A/m-RNA methylation in stress response regulation
N6-Methyladenosine (m6A) and N6,2'-O-dimethyladenosine (m6Am) are abundant mRNA modifications that regulate transcript processing and translation. The role of both, here termed m6A/m, in the stress re
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